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IκB-α (Phospho Ser32) Rabbit mAb

商品貨號： PRM8715

適應性： 人,小鼠,大鼠

￥800元

規(guī)格：

50ul

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說明書

商品描述

發(fā)貨日期：現(xiàn)貨
靶點-Target： IκB α
反應性-Reactivity：人,小鼠,大鼠
應用-Applications： WB,IF,IP,ELISA
MW(Calculated)： 36kD
MW(Observed)： 40kD
宿主物種-Host Species： Rabbit
同種型-Isotype： IgG,Kappa
偶聯(lián)-Conjugate： Phospho
修飾-Modification： Ser32
推薦稀釋比： WB 1:2000-1:10000;IF 1:200-1:1000;ELISA 1:5000-1:20000;IP 1:50-1:200;
組成： PBS, 50% glycerol, 0.05% Proclin 300, 0.05%BSA
純化工藝： Protein A
儲存： -15°C to -25°C/1 year(Do not lower than -25°C)
克隆號： PT0899R
特異性： IκB-α (Phospho Ser32) antibody detects endogenous levels of IκB-α only when phosphorylated at Ser32.and dually phosphorylated at two sites.The name of modified sites may be influenced by many factors, such as species (the modified site was not originally found in human samples) and the change of protein sequence (the previous protein sequence is incomplete, and the protein sequence may be prolonged with the development of protein sequencing technology). When naming, we will use the "numbers" in historical reference to keep the sites consistent with the reports. The antibody binds to the following modification sequence (lowercase letters are modification sites):HDsGL
基因名稱： NFKBIA IKBA MAD3 NFKBI
蛋白名稱： NF-kappa-B inhibitor alpha
別名： NF-kappa-B inhibitor alpha;I-kappa-B-alpha;IkB-alpha;IkappaBalpha;Major histocompatibility complex enhancer-binding protein MAD3;
Organism-1： Human
基因ID-1： 4792
SwissProt-1： P25963
Organism-2： Mouse
基因ID-2： 18035
SwissProt-2： Q9Z1E3
Organism-3： Rat
基因ID-3： 25493
SwissProt-3： Q63746
背景： This gene encodes a member of the NF-kappa-B inhibitor family, which contain multiple ankrin repeat domains. The encoded protein interacts with REL dimers to inhibit NF-kappa-B/REL complexes which are involved in inflammatory responses. The encoded protein moves between the cytoplasm and the nucleus via a nuclear localization signal and CRM1-mediated nuclear export. Mutations in this gene have been found in ectodermal dysplasia anhidrotic with T-cell immunodeficiency autosomal dominant disease. [provided by RefSeq, Aug 2011],
細胞定位： Cytoplasm. Nucleus. Shuttles between the nucleus and the cytoplasm by a nuclear localization signal (NLS) and a CRM1-dependent nuclear export. .

IκB-α (Phospho Ser32) Rabbit mAb

Catalog No PRM8715

Product information

發(fā)貨日期：現(xiàn)貨
靶點-Target： IκB α
反應性-Reactivity：人,小鼠,大鼠
應用-Applications： WB,IF,IP,ELISA
MW(Calculated)： 36kD
MW(Observed)： 40kD
宿主物種-Host Species： Rabbit
同種型-Isotype： IgG,Kappa
偶聯(lián)-Conjugate： Phospho
修飾-Modification： Ser32
推薦稀釋比： WB 1:2000-1:10000;IF 1:200-1:1000;ELISA 1:5000-1:20000;IP 1:50-1:200;
組成： PBS, 50% glycerol, 0.05% Proclin 300, 0.05%BSA
純化工藝： Protein A
儲存： -15°C to -25°C/1 year(Do not lower than -25°C)
克隆號： PT0899R
特異性： IκB-α (Phospho Ser32) antibody detects endogenous levels of IκB-α only when phosphorylated at Ser32.and dually phosphorylated at two sites.The name of modified sites may be influenced by many factors, such as species (the modified site was not originally found in human samples) and the change of protein sequence (the previous protein sequence is incomplete, and the protein sequence may be prolonged with the development of protein sequencing technology). When naming, we will use the "numbers" in historical reference to keep the sites consistent with the reports. The antibody binds to the following modification sequence (lowercase letters are modification sites):HDsGL
基因名稱： NFKBIA IKBA MAD3 NFKBI
蛋白名稱： NF-kappa-B inhibitor alpha
別名： NF-kappa-B inhibitor alpha;I-kappa-B-alpha;IkB-alpha;IkappaBalpha;Major histocompatibility complex enhancer-binding protein MAD3;
Organism-1： Human
基因ID-1： 4792
SwissProt-1： P25963
Organism-2： Mouse
基因ID-2： 18035
SwissProt-2： Q9Z1E3
Organism-3： Rat
基因ID-3： 25493
SwissProt-3： Q63746
背景： This gene encodes a member of the NF-kappa-B inhibitor family, which contain multiple ankrin repeat domains. The encoded protein interacts with REL dimers to inhibit NF-kappa-B/REL complexes which are involved in inflammatory responses. The encoded protein moves between the cytoplasm and the nucleus via a nuclear localization signal and CRM1-mediated nuclear export. Mutations in this gene have been found in ectodermal dysplasia anhidrotic with T-cell immunodeficiency autosomal dominant disease. [provided by RefSeq, Aug 2011],
細胞定位： Cytoplasm. Nucleus. Shuttles between the nucleus and the cytoplasm by a nuclear localization signal (NLS) and a CRM1-dependent nuclear export. .

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